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Several studies have demonstrated that a modest reduction of body weight of 5-10% can help to substantially and rapidly improve the CAD and type 2 diabetes risk profiles of viscerally obese patients independent of notable regression of coronary stenosis.
Weight loss in abdominally obese promotes selective mobilization of visceral adipose tissue with a ~10% weight loss resulting in ~30% decrease in visceral adipose tissue regardless of the weight loss strategy (diet, exercise, drug).
The selective mobilization of VAT leads to improvement in lipid profile (lower TG and Apo-B and raises HDL), glycemic control, blood pressure and variables of endothelial dysfunction, fibrinolysisand chronic inflammation thus reducing the risk of thrombosis.
These synergistic, metabolic improvements may help stabilize unstable atheromatous plaque in the viscerally obese, thereby reducing the risk of acute coronary events.

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Hypertriglyceridemic Waist is the syndrome associated with the metabolic triad described previously.
At present, physicians readily treat the complications associated with the syndrome and obesity in general, but rarely attack the cause.
Weight management for the overweight and obese - and especially the viscerally obese - is a necessary component of primary and secondary prevention of CHD.